南京林业大学学报(自然科学版) ›› 1990, Vol. 33 ›› Issue (04): 8-13.doi: 10.3969/j.jssn.1000-2006.1990.04.003

• 研究论文 • 上一篇    下一篇

卅烷醇对湿地松幼苗的生理效应

俞德超;汪安琳   

  1. 南京林业大学林学系;中科院植物生理研究所博士生;南京林业大学林学系
  • 出版日期:1990-12-18 发布日期:1990-08-18

THE PHYSIOLOGICAL EFFECTS OF TRIACONTANOL (TA) ON PINUS ELLIOTTH ENGELM. SEEDLINGS

Yu Dechao Wang Anlin   

  1. Nanjing Forestry University
  • Online:1990-12-18 Published:1990-08-18

摘要: <正>卅烷醇(TA)0.5和1.0ppm对不同苗龄湿地松幼苗硝酸还原酶(NR)活力诱导均有促进作用。TA不能单独诱导NR活力,但它能促进NO_3~-对NR的诱导。TA能部分抵消砷酸钠对NR活力的抑制。TA促进黄化苗叶绿素的积累紧密平行于它对NR诱导的促进。CHM对TA促进黄化针叶及根的NR诱导有明显的抑制作用,而LCMC只抑制TA对针叶的效应,不抑制TA对离体根的作用。经TA处理的湿地松幼苗,其高生长、根系生长及各器官的生物量均显著高于对照。

Abstract: TA could stimulate NK activity induction by KNO3 in Pinus elliottii Engelm. seedlings at different ages. TA could not induce NR activity alone, but could enhance NR induction by NO3. TA eliminated partially the inhibition of arsenate (an oxidate phosphorylation decoupler). The accumulation of chlorophyll accelerated by TA or BA closely paralleled to the increase of NRA accelerated by TA or BA. Cycloheximide (CHM) could inhibit the effect of TA on NRA in roots and excised etiolated leaves exposed to light and NO3. Lincomycin (LCMC, an inhibitor of chloroplast protein synthesis) could only inhibit the effect of TA in etiolated leaves, but did not inhibit the effect of TA on NRA in roots. The inhibition of CHM was much stronger than LCMC.TA(0.5ppm, l.0ppm) could increase the height of seedlings in the field experiment. The root system of TA treated seedlings was superior to the control.